By Lars Mecklenburg, Monika Linek, Desmond J. Tobin
Hair Loss issues in family Animals is an in-depth reference at the pathomechanisms and medical ways of all dermis illnesses in household animals that experience hair loss because the important medical function. It provides either simple and clinically-relevant wisdom on alopecic disorder in animals. this article is a different source supplying state of the art insurance of the body structure and pathology of hair follicles. Disease-specific chapters comprise: a close description of the sickness entity, etiology, pathogenesis, scientific and histopathological analysis and remedy modalities.
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Additional resources for Hair Loss Disorders in Domestic Animals
Example text
1991a). 2 Hair follicle stem cells The highly regenerative and highly differentiated hair follicle tissue has prompted several researchers to suggest that the hair follicle may be exploited as a repository of primitive cell populations for replacement in other tissues and organs. 3f,g). Despite much recent progress in dissecting the main events driving the continual hair cycling that form the basis of a fairly robust hypothesis, we still are some way from explaining some critical aspects of the hair growth cycle including its intrinsic periodicity, how 23 autonomy continues to reside in single hair follicles, and how cycling persists throughout adult life.
Epi-Mc, epidermal melanocyte; IF-Mc, infundibular melanocyte; SG, sebaceous gland; ORS-AMc, outer root sheath amelanogenic melanocyte; FDP, follicular dermal papilla; B-MMc, bulbar melanogenic melanocyte; B-AMc, bulbar amelanotic melanocyte; HS, hair shaft. The cartoon also depicts perifollicular blood vessels, nerves and mast cells. ) 28 Hair follicle biology hair bulb next to the follicular papilla. Melanogenic bulbar melanocytes interact closely with the follicular papilla via the thin basal lamina that separates them from the mesenchymal follicular papilla (Fig.
The biosynthesis of hair proteins begins in the bulb of the anagen hair follicle and ceases in the keratogeneous zone, approximately 500 µm above the level of the zone of maximal keratinocyte proliferation (so-called sub-Auber’s matrix) in human scalp terminal hair follicles. This location will differ of course for hair follicles in other regions of the human body, and also for other mammalian species. Low-sulfur and high-sulfur proteins are synthesized simultaneously, though the synthesis of the latter peaks later.