By J. De Groote (auth.), P. Fondu MD, O. Thijs MD (eds.)
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Verstraete Do you have any speculation on the continuously counter wich, deprived from the early phase of Coumarine therapy and could you explain if it has something to do with a drop of Factor VII? Mannucci That is an interesting suggestion, I don't know but it's a very interesting idea. Question from the audience Mannucci I cannot answer, the only thing I can tell you is that in our group, in our hands as much as there was a proportional decrease of Protein C in the progressive forms, I mean more in chronic active and chronic persistance and more in cirrhosis than in chronic active this was also true for other indexes of liver synthetic capacity, that is albumin, choline-esterase and prothrombin time.
Hauschka PV 1980. Carboxylated calcium binding proteins and vitamin K. N. Engl. J. Med. 302:1460-1466. 11. Nishimoto SK, Price PA. 1980. Secretion of the vitamin K-dependent protein of bone by rat osteosarcoma cells. J. Biol. Chern. 255:6579-6583. 47 12. Tomita A. 1971. Post menopausal osteoporosis Ca study with vitamin K2 • Clin. Endocrinol. 19:731-736. 35 13. Lian JB, Prien EL Jr, Glimcher MJ, Gallop PM. 1977. The presence of protein-bound gamma-carboxyglutamic acid in calcium-containing renal calculi.
They suggested that this potent anticoagulant activity, called autoprothrombin II-A, was derived from prothrombin that had been activated by limited thrombin proteolysis. A decade later, Marciniak (8) suggested that human autoprothrombin II-A was not derived from prothrombin but was a distinct species-specific protein. The anticoagulant activity of activated bovine Protein C was initially ascribed to a competitive inhibition of factor X (9, 10). a 37 Subsequently, various investigators have shown that bovine activated protein C enzymatically inactivates bovine factors V and VIII by limited proteolysis (10, 11, 12).